Idiopathic facial aseptic granuloma
The pathogenesis of IFAG is unknown, and no common predisposing factors including family history or prior trauma have been consistently associated. It has been hypothesized that IFAG may be due to a reactive process around an embryological remnant or may be related to granulomatous rosacea given its association with chalazia.
L92.9 – Granulomatous disorder of the skin and subcutaneous tissue, unspecified
123633001 – Focal granulomatous inflammation
- Pilomatricoma – Unlike IFAG, pilomatricoma has a positive teeter-totter sign and is firmer in texture.
- Dermoid and epidermoid cysts
- Bacterial pyoderma (abscess, furuncle) – Culture for bacteria in IFAG remains negative except in cases of superinfection, and IFAG is painless in comparison.
- Atypical mycobacterial infection
- Cutaneous leishmaniasis (New World, Old World)
- Fungal (sporotrichosis, blastomycosis, cryptococcosis, coccidioidomycosis)